Renodapt S 360 mg consists of the active ingredient mycophenolate sodium, which is an immunosuppressive agent. It is used to prevent rejection of organs in kidney transplant procedures by suppressing the excess immune reaction against the newly transplanted organ.
Prevention of Kidney Transplant Rejection
During organ transplant procedures, there is a risk of graft rejection, in which the host’s immune system identifies the newly transplanted organ as “non-self” and activates a series of immune cascade reactions, which act against the organ. This response can be in the form of antibodies, a delayed cell-mediated response, or chronic rejection of the organ by the immune system. This results in an inflammatory response and tissue damage to the organ. It also causes progressive damage to the graft and, finally, complete graft (organ) failure, if the condition is left untreated.
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The dosage of Renodapt S 360 mg is based on the patient’s Body Surface Area (BSA) and is calculated in mg/m² for pediatric dosing.
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After an organ transplant procedure, the patient's immune system of the patient recognizes the human leukocytes (HLA), which are a group of surface antigens on the donor tissue, as foreign, which causes the T-lymphocyte response to be activated through a process called antigen presentation to the antigen-presenting cells (APCs), and also provide costimulation by secreting cytokines (IL-2).
Once activated, the T-cells proliferate and differentiate into cytotoxic T-cells, which are capable of causing damage to graft tissue. Also, the activated helper T-cells stimulate B-lymphocytes to produce antibodies directed against the HLA antigens present on the transplanted organ. This combined cellular and humoral immunologic response produces inflammation and eventually damages the graft endothelium, which ultimately will result in functional failure of the organ.
Renodapt S 360 mg (Mycophenolate sodium) works by suppressing this immunologic response via the inhibition of the enzyme inosine monophosphate dehydrogenase (IMPDH). This enzyme is essential for the de novo synthesis of guanine nucleotides. Both T and B lymphocytes depend substantially on this pathway for their DNA synthesis, as these cells cannot utilize salvage pathways like other cells. The inhibition of IMPDH will deplete the levels of guanosine nucleotides necessary for the proliferation of T or B-lymphocytes.
By selectively suppressing the activity of T and B lymphocytes, Renodapt S 360 mg prevents the immune-mediated destruction of the transplanted organ and protects the structure and function of the graft.
Common side effects
Effects due to immunosuppression
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1.What is Renodapt S 360 mg used for?
Renodapt S 360 mg is used to prevent kidney transplant rejections.
2.What are the critical symptoms?
Critical symptoms include high fever, gastrointestinal symptoms, and sudden shortness of breath.
3.Can I stop taking the drug if I feel better?
Patients must complete the entire prescribed course of the medication.
4.Can breastfeeding women take Renodapt S 360 mg?
This drug must not be taken by lactating women as it can pass into the breastmilk.
5.How does this drug work?
Renodapt S 360 mg works by suppressing the excess immune response.
